The issue at hand here is not so much the acceptability of incest and bestiality but the fact that they are mile markers on the slippery slope that was predicted at the begining of the debate on same sex marriage. The fact that these groups (and others) are now making their case for inclusion in the accepted behavior club confirms that validity of the slippery slope argument. They are clinging to the coat tails of the same sex marriage movement by using the same arguments.
http://www.consang.net/images/d/dd/01AHBWeb3.pdfAnd, while the Rothchilds may have escaped the consequences of inbreeding (according to that one article), other European dynasts were not so lucky:
Consanguinity, morbidity, and mortality
The detrimental health effects associated with consanguinity are caused by the expression of rare, recessive genes inherited from a common ancestor(s). In populations where inbred unions are common, increased levels of morbidity and mortality caused by the action of detrimental recessive genes can be predicted. Generally, inbreeding is associated with loss of biological fitness. It is however appropriate to note that, even in the absence of preferential consanguinity, alleles which are rare in large populations can rapidly increase to high frequency in a breeding pool of restricted size, because of factors such as founder effect and random genetic drift.
Empirical studies on the progeny of first cousins indicate morbidity levels to be some 1% to 4% higher than in the offspring of unrelated couples (reviewed in Bittles and Makov 1988). The less common a disorder, the greater the influence of consanguinity on its prevalence, a generalization that applies to recessive multigene disorders as well as to single gene conditions. For this reason, many previously unrecognized genetic diseases have first been diagnosed in highly endogamous communities, and in a significant proportion of cases the underlying mutation may be unique to the community. At a practical level, this community-specific pattern of disease leads to major problems when attempting to estimate
the burden imposed by consanguinity-associated morbidity at national or even at regional and local levels.
In a study based on combined data from 38 populations in eastern and southern Asia, the Middle East, Africa, Europe, and South America, with average coefficient of inbreeding (α) values ranging from 0.0005 to 0.0370, mean excess mortality at the first cousin level was 4.4% (Bittles and Neel 1994). This estimate appears to be valid for all of the large human populations so far examined. However, consanguinity interacts with a range of sociodemographic variables in determining rates of mortality during infancy and early childhood. When these influences were simultaneously analyzed using data collected retrospectively as part of the 1990/91 Pakistan Demographic and Health Survey, the major determinants of early death were maternal illiteracy, maternal age at birth of less than 20 years, and a birth interval of less than 18 months. But, even after controlling for these factors, first cousin progeny had statistically significant odds ratios for neonatal, postneonatal, and infant mortality of 1.36, 1.28, and 1.32, respectively (Grant and Bittles 1997).
The Role of Inbreeding in the Extinction of a European Royal DynastyIn other words, there are numerous medical reasons for the incest taboos and laws.
The kings of the Spanish Habsburg dynasty (1516–1700) frequently married close relatives in such a way that uncle-niece, first cousins and other consanguineous unions were prevalent in that dynasty. In the historical literature, it has been suggested that inbreeding was a major cause responsible for the extinction of the dynasty when the king Charles II, physically and mentally disabled, died in 1700 and no children were born from his two marriages, but this hypothesis has not been examined from a genetic perspective. In this article, this hypothesis is checked by computing the inbreeding coefficient (F) of the Spanish Habsburg kings from an extended pedigree up to 16 generations in depth and involving more than 3,000 individuals. The inbreeding coefficient of the Spanish Habsburg kings increased strongly along generations from 0.025 for king Philip I, the founder of the dynasty, to 0.254 for Charles II and several members of the dynasty had inbreeding coefficients higher than 0.20. In addition to inbreeding due to unions between close relatives, ancestral inbreeding from multiple remote ancestors makes a substantial contribution to the inbreeding coefficient of most kings. A statistically significant inbreeding depression for survival to 10 years is detected in the progenies of the Spanish Habsburg kings. The results indicate that inbreeding at the level of first cousin (F = 0.0625) exerted an adverse effect on survival of 17.8%±12.3. It is speculated that the simultaneous occurrence in Charles II (F = 0.254) of two different genetic disorders: combined pituitary hormone deficiency and distal renal tubular acidosis, determined by recessive alleles at two unlinked loci, could explain most of the complex clinical profile of this king, including his impotence/infertility which in last instance led to the extinction of the dynasty.